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Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1997 1
2000 2
2002 4
2003 4
2004 5
2005 5
2006 6
2007 6
2008 2
2009 3
2010 3
2011 3
2012 6
2013 1
2014 3
2015 3
2016 4
2017 6
2018 3
2019 3
2020 1
2021 3
2022 3
2024 1

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71 results

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Page 1
Acetyl-Coenzyme A Synthetase 2 Potentiates Macropinocytosis and Muscle Wasting Through Metabolic Reprogramming in Pancreatic Cancer.
Zhou Z, Ren Y, Yang J, Liu M, Shi X, Luo W, Fung KM, Xu C, Bronze MS, Zhang Y, Houchen CW, Li M. Zhou Z, et al. Gastroenterology. 2022 Nov;163(5):1281-1293.e1. doi: 10.1053/j.gastro.2022.06.058. Epub 2022 Jun 28. Gastroenterology. 2022. PMID: 35777482 Free PMC article.
In this study, we demonstrate that ACSS2 potentiates macropinocytosis and muscle wasting through metabolic reprogramming in pancreatic cancer. ...ZIP4 induces macropinocytosis through cyclic adenosine monophosphate response element-binding protein-activated syndecan 1 (SDC …
In this study, we demonstrate that ACSS2 potentiates macropinocytosis and muscle wasting through metabolic reprogramming in pancreati …
Circulating Monocytes Act as a Common Trigger for the Calcification Paradox of Osteoporosis and Carotid Atherosclerosis via TGFB1-SP1 and TNFSF10-NFKB1 Axis.
Zeng Z, Guo R, Wang Z, Yan H, Lv X, Zhao Q, Jiang X, Zhang C, Zhang D, Yang C, Li W, Zhang Z, Wang Q, Huang R, Li B, Hu X, Gao L. Zeng Z, et al. Front Endocrinol (Lausanne). 2022 Jul 22;13:944751. doi: 10.3389/fendo.2022.944751. eCollection 2022. Front Endocrinol (Lausanne). 2022. PMID: 35937796 Free PMC article.
Then, ligand-receptor interactions between CD14(+) monocytes and osteoblast and vascular smooth muscle cell (SMC) lineages were annotated with iTALK. Finally, we selected calcification paradox-related expression in circulating monocytes with LASSO analysis. ...Second, CD14 …
Then, ligand-receptor interactions between CD14(+) monocytes and osteoblast and vascular smooth muscle cell (SMC) lineages were annot …
Cardioprotection of M2 macrophages-derived exosomal microRNA-24-3p/Tnfsf10 axis against myocardial injury after sepsis.
Sun X, Liu Y, Wang J, Zhang M, Wang M. Sun X, et al. Mol Immunol. 2022 Jan;141:309-317. doi: 10.1016/j.molimm.2021.11.003. Epub 2021 Dec 18. Mol Immunol. 2022. PMID: 34933177 Free article.
RESULTS: miR-24-3p expression dropped while Tnfsf10 expression raised in the myocardial tissue of septic mice. ...A binding relation exhibited between miR-24-3p and Tnfsf10, and M2-exo-derived miR-24-3p alleviated LPS-induced myocardial injury by inhibiting Tnfsf …
RESULTS: miR-24-3p expression dropped while Tnfsf10 expression raised in the myocardial tissue of septic mice. ...A binding relation …
TRAIL in the airways.
Robertson NM, Rosemiller M, Lindemeyer RG, Steplewski A, Zangrilli JG, Litwack G. Robertson NM, et al. Vitam Horm. 2004;67:149-67. doi: 10.1016/S0083-6729(04)67009-9. Vitam Horm. 2004. PMID: 15110176 Review.
TRAIL is expressed in the airways by inflammatory cells infiltrated in the bronchial mucosa, as well as by structural cells of the airway wall including fibroblasts, epithelial, endothelial, and smooth muscle cells. By releasing TRAIL, these different cell types may then p …
TRAIL is expressed in the airways by inflammatory cells infiltrated in the bronchial mucosa, as well as by structural cells of the airway wa …
Role of mitochondrial function in cell death and body metabolism.
Lee MS. Lee MS. Front Biosci (Landmark Ed). 2016 Jun 1;21(6):1233-44. doi: 10.2741/4453. Front Biosci (Landmark Ed). 2016. PMID: 27100503 Free article. Review.
Mitochondrial DNA (mtDNA)-depleted r0 cells were resistant to diverse apoptosis inducers such as TNF-alpha, TNFSF10, staurosporine and p53. Apoptosis resistance was accompanied by the absence of mitochondrial potential loss or cytochrome c translocation. r0 cells were also …
Mitochondrial DNA (mtDNA)-depleted r0 cells were resistant to diverse apoptosis inducers such as TNF-alpha, TNFSF10, staurosporine an …
Expression of apoptosis related proteins in normal and diseased muscle: a possible role for Bcl-2 in protection of striated muscle.
Danielsson O, Nilsson C, Lindvall B, Ernerudh J. Danielsson O, et al. Neuromuscul Disord. 2009 Jun;19(6):412-7. doi: 10.1016/j.nmd.2009.03.008. Epub 2009 May 23. Neuromuscul Disord. 2009. PMID: 19477129
The unique absence of major histocompatibility complex class I antigen (MHC-I) expression in normal muscle is one possible mechanism protecting striated muscle. In order to define their possible involvement in protection of normal muscle, we investigated the …
The unique absence of major histocompatibility complex class I antigen (MHC-I) expression in normal muscle is one possible mechanism …
T cells and eosinophils in bronchial smooth muscle cell death in asthma.
Solarewicz-Madejek K, Basinski TM, Crameri R, Akdis M, Akkaya A, Blaser K, Rabe KF, Akdis CA, Jutel M. Solarewicz-Madejek K, et al. Clin Exp Allergy. 2009 Jun;39(6):845-55. doi: 10.1111/j.1365-2222.2009.03244.x. Epub 2009 Apr 14. Clin Exp Allergy. 2009. PMID: 19400895
BACKGROUND: Bronchial smooth muscle cells (SMC) proliferate, express adhesion molecules, secrete cytokines and thus efficiently contribute to the pathogenesis of asthma. ...
BACKGROUND: Bronchial smooth muscle cells (SMC) proliferate, express adhesion molecules, secrete cytokines and thus efficiently contr …
Muscle-derived TRAIL negatively regulates myogenic differentiation.
Kim D, Singh N, Waldemer-Streyer RJ, Yoon MS, Chen J. Kim D, et al. Exp Cell Res. 2020 Sep 1;394(1):112165. doi: 10.1016/j.yexcr.2020.112165. Epub 2020 Jul 6. Exp Cell Res. 2020. PMID: 32645396 Free PMC article.
TNF-related apoptosis-inducing ligand (TRAIL) is known to induce apoptosis in cancer cells, although non-apoptotic functions have also been reported for this cytokine in various cell types. TRAIL and its receptor TRAIL-R2 are expressed in skeletal muscles, but a potential …
TNF-related apoptosis-inducing ligand (TRAIL) is known to induce apoptosis in cancer cells, although non-apoptotic functions have also been …
Trail and vascular injury.
Martin-Ventura JL, Munoz-Garcia B, Egido J, Blanco-Colio LM. Martin-Ventura JL, et al. Front Biosci. 2007 May 1;12:3656-67. doi: 10.2741/2342. Front Biosci. 2007. PMID: 17485329 Review.
The main mechanisms involved in atherogenesis are: lipoprotein retention, endothelial cell activation, vascular smooth muscle cell proliferation, macrophage infiltration, proteolytic injury, neovascularization and apoptosis. ...
The main mechanisms involved in atherogenesis are: lipoprotein retention, endothelial cell activation, vascular smooth muscle cell pr …
Effects of prostaglandin analogues on human ciliary muscle and trabecular meshwork cells.
Zhao X, Pearson KE, Stephan DA, Russell P. Zhao X, et al. Invest Ophthalmol Vis Sci. 2003 May;44(5):1945-52. doi: 10.1167/iovs.02-0920. Invest Ophthalmol Vis Sci. 2003. PMID: 12714628
Aquaporin-1 and versican were downregulated in the HCM, whereas IGF1 and fibroleukin were upregulated in HTM. Expression levels of TNFSF10 and promelanosome-concentrating hormone also increased in the treated HTM cells. The mRNA levels for the prostaglandin FP receptor wer …
Aquaporin-1 and versican were downregulated in the HCM, whereas IGF1 and fibroleukin were upregulated in HTM. Expression levels of TNFSF1 …
71 results